Jahahreeh "Jay" Finley focuses his practice on civil litigation and is a registered United States Patent and Trademark Office patent attorney. He has experience supporting pharmaceutical, biotechnology, healthcare, and technology clients in matters involving patent disputes, antitrust investigations, regulatory compliance, and corporate transactions.

Jay’s work has included advising on intellectual property portfolio management, freedom-to-operate analyses, and healthcare regulations. He also has experience supporting high-stakes litigation, managing discovery, and coordinating with in-house legal teams and outside counsel.

Before entering the legal profession, Jay conducted scientific research in biotechnology and has continued to contribute to the field as an inventor and published author. He is the named inventor on a U.S. patent and has authored multiple peer-reviewed publications focused on biotechnology, metabolic disease, HIV, and related scientific research.

Jay earned his J.D., with a concentration in Health Law, from the University of Houston Law Center, where he received a Writing Award. He also earned an M.S. in Biotechnology from The Johns Hopkins University, an M.B.A., summa cum laude, from Alabama A&M University, and a B.S. in Biology, magna cum laude, from Alabama A&M University. He is admitted to practice in Texas and is a registered patent attorney before the United States Patent and Trademark Office.

Finley J. Cellular stress and AMPK links metformin and diverse compounds with accelerated emergence from anesthesia and potential recovery from disorders of consciousness. Med Hypotheses. 2019 Mar:124:42-52.

Finley J. Transposable elements, placental development, and oocyte activation: cellular stress and AMPK links jumping genes with the creation of human life. Med Hypotheses. 2018 Sep:118:44-54.

Finley J. Cellular stress and AMPK activation as a common mechanism of action linking the effects of metformin and diverse compounds that alleviate accelerated aging defects in Hutchinson-Gilford progeria syndrome. Med Hypotheses. 2018 Sep:118:151-162.

Finley J. Facilitation of hippocampal long-term potentiation and reactivation of latent HIV-1 via AMPK activation: common mechanism of action linking learning, memory, and the potential eradication of HIV-1. Med Hypotheses. 2018 Jul:116:61-73.

Finley J. Elimination of cancer stem cells and reactivation of latent HIV-1 via AMPK activation: common mechanism of action linking inhibition of tumorigenesis and the potential eradication of HIV-1. Med Hypotheses. 2017 Jul:104:133-146.

Finley J. Oocyte activation and latent HIV-1 reactivation: AMPK as a common mechanism of action linking the beginnings of life and the potential eradication of HIV-1. Med Hypotheses. 2016 Aug:93:34-47.

Finley J. Reactivation of latently infected HIV-1 viral reservoirs and correction of aberrant alternative splicing in the LMNA gene via AMPK activation: Common mechanism of action linking HIV-1 latency and Hutchinson-Gilford progeria syndrome. Med Hypotheses. 2015 Sep;85(3):320-32.

Finley J. Alteration of splice site selection in the LMNA gene and inhibition of progerin production via AMPK activation. Med Hypotheses. 2014 Nov;83(5):580-7.